Protein metabolism following injury.

Protein metabolism following injury.

Fig. 1 Comparisons in two burned patients between the rate of catabolism ofplasma albumin and y G globulin (as glkg body weight) and the basal metabolic rate (BMR) as percentages above the expected normal value. The patient treated in the cool environment had a burned area covering 30% of the body surface, 10% being full-thickness skin loss; the patient treated in the warm environment had a bur...

Protein Metabolism after Injury.

Ballantyne, F. C., Cuthbertson, D. P., Fleck, A. & Tilstone, W. J. (1969). Int. Congr. Nutr. VIII Pru,rpie, 1969. Barr, P.-O., Birke, G., Liljedahl, S.-0. & Plantin, L.-0. (1968). Lancet i, 164. Caldwell, F. T. (1962). Ann. Surg. 155, 119. Caldwell, I;. T. (1970). In Energy Metabolism in Trauma, Ciba Foundation Symposium, p. 23. London: Churchill. Cuthbertson, D. P. (1970). BY.^. Surg. 57, 718....

Proteose Intoxications and Injury of Body Protein. I. T~e Metabolism of Fasting Dogs following Proteose

Hyperoxia results in increased aerobic metabolism following acute brain injury

Acute brain injury is associated with depressed aerobic metabolism. Below a critical mitochondrial pO2 cytochrome c oxidase, the terminal electron acceptor in the mitochondrial respiratory chain, fails to sustain oxidative phosphorylation. After acute brain injury, this ischaemic threshold might be shifted into apparently normal levels of tissue oxygenation. We investigated the oxygen dependenc...

Mitogen-Activated Protein Kinase Pathways Following Traumatic Brain Injury

The mechanisms underlying the secondary or delayed cell death in the hippocampus and cerebral hemisphere after traumatic brain injury (TBI) have been poorly understood. Recent data suggesting that TBI may have relationship with both an inflammatory and a neurodegenerative factors are also presented. Mitogen-activated protein kinases (MAPK), which play a crucial role in signal transduction, are ...